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6th Internet World Congress for Biomedical Sciences

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SCALP NECROSIS AND HERPES ZOSTER OPHTALMICUS

Alejandro Nogueira(1), Olatz Alcelay(2), MŞ Jesús Martínez González(3), Teresa Pérez (4)
(1)(2)(3)(4)Hospital Central de Asturias - Oviedo. Spain

[ABSTRACT] [INTRODUCTION] [MATERIAL & METHODS] [RESULTS] [IMAGES] [DISCUSSION] [CONCLUSIONS] [BIBLIOGRAPHY] [Discussion Board]
INTRODUCTION Previous: Age and regional peculiarities of the microbe landscape of the nasal mucosa.
[Infectious Diseases]
Previous: The Difference of the Ocular Blood Circulation Between Normal-Tension Glaucomas and Normal Controls.
[Ophthalmology]
Previous: RECONSTRUCTION OF NASAL AMPUTATION BY HUMAN BITE RESULTS
[Plastic Surgery]
Next: NEURILEMOMAS OR SCHWANNOMAS OF THE UPPER EXTREMITY 
(REVISION OF A 5-YEARS PERIOD)

MATERIAL & METHODS

Case 1: 78 year-old, male, with antecedent of previous left senile catharat, presents right trigeminal herpes zoster affecting ophtalmic and maxilar-trigeminal facial skin. Cutaneous procces is extended to the right orbicular and malar zone presenting alveolar inflamation of upper right maxila without teeth loss. It´s resoluted in 15 days.

At 8th day the patient suffers blindness of right eye during 5 days that heals without treatment. One month later he develops nacrotic plaque, 15x10 cm, on right temporal and bilateral parieto-frontal area. Ophtalmic and radiologic studies are normal. Right carotid arteriography results in stenosis of 50% at the external carotid origin, stop at exit of internal maxilar and superficial temporal artery thrombosis. Likewise rich colateral circulation is developed. Lumen of internal carotid artery presents abundant irregularities with very thin wall of ophtalmic division. Superficial temporal artery biopsy presents trombosed with fibroconnective tissue inside, intimate is thick and muscular presents focal inflammatory infiltration without giant cells does not extend to other level.

It´s debrided necrosis plaque, exposing periosteal loss bone. It´s removed external table and is applied split thickness skin grafts that took partiallity. It´s neccesary two times more for ostectomyes and split thickness skin grafts again until reepitelitation completely. Not cantral neurologic complication appears.

Case 2: 67 year-old, male, at cronic corticosteroid treatment, debuts with left ophtalmic herpes, acute retinian necrosis, oculomotor palsy, queratitis and blidness, simultaneous un days. Antiviral treatment is given. The following month he developed headache, right hemiplegy, level conscience decreased and heé taken to intensive care unit. At that moment he presented left scalp necrosis that after debridement, frontal lobe is exposed in 10x8 cm area. Arteriography proves superficial temporal and central retinal artery thrombosis. There were irregularities at left intracraneal vessels with multiples aneurismatic changes and hemorrhagic at internal carotid system.

The patient suffers neurologic damage progresively until death 6 weeks later.


Discussion Board
Discussion Board

Any Comment to this presentation?

[ABSTRACT] [INTRODUCTION] [MATERIAL & METHODS] [RESULTS] [IMAGES] [DISCUSSION] [CONCLUSIONS] [BIBLIOGRAPHY] [Discussion Board]

INTRODUCTION Previous: Age and regional peculiarities of the microbe landscape of the nasal mucosa.
[Infectious Diseases]
Previous: The Difference of the Ocular Blood Circulation Between Normal-Tension Glaucomas and Normal Controls.
[Ophthalmology]
Previous: RECONSTRUCTION OF NASAL AMPUTATION BY HUMAN BITE RESULTS
[Plastic Surgery]
Next: NEURILEMOMAS OR SCHWANNOMAS OF THE UPPER EXTREMITY 
(REVISION OF A 5-YEARS PERIOD)
Alejandro Nogueira, Olatz Alcelay, MŞ Jesús Martínez González, Teresa Pérez
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Last update: 15/01/00